Bax overexpression enhances cytochrome c release from mitochondria and sensitizes KATOIII gastric cancer cells to chemotherapeutic agent-induced apoptosis.

International journal of oncology

Volumn 16, Issue 4, 2000, Pages 745-749

  Sawa, H ^a   Kobayashi, T ^a   Mukai, K ^a   Zhang, W ^a   Shiku, H ^a  

^a MIE UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

ANTINEOPLASTIC AGENT; BAX PROTEIN, HUMAN; BENZYLOXYCARBONYLVALYL ALANYL ASPARTYL FLUOROMETHYL KETONE; BENZYLOXYCARBONYLVALYL-ALANYL-ASPARTYL FLUOROMETHYL KETONE; CYTOCHROME C; ONCOPROTEIN; PEPTIDE CHLOROMETHYL KETONE; PROTEIN BAX; PROTEIN BCL 2;

APOPTOSIS; ARTICLE; CELL CULTURE; DRUG EFFECT; ENZYMOLOGY; HUMAN; MITOCHONDRION; PATHOLOGY; PHYSIOLOGY; SECRETION; STOMACH TUMOR;

AMINO ACID CHLOROMETHYL KETONES; ANTINEOPLASTIC AGENTS; APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; CYTOCHROME C GROUP; HUMANS; MITOCHONDRIA; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; STOMACH NEOPLASMS; TUMOR CELLS, CULTURED;

EID: 0034164496     PISSN: 10196439     EISSN: None     Source Type: Journal    
DOI: 10.3892/ijo.16.4.745     Document Type: Article

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