Mutational analysis of the CTNNB1 (β-catenin) gene in human endometrial cancer: Frequent mutations at codon 34 that cause nuclear accumulation

Oncology Reports

Volumn 7, Issue 2, 2000, Pages 323-326

  Horii, Akira ^a  

^a TOHOKU UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

catenin; CTNNB1; Endometrial cancer; Somatic mutation

Indexed keywords

BETA CATENIN; CTNNB1 PROTEIN, HUMAN; CYTOSKELETON PROTEIN; TRANSACTIVATOR PROTEIN;

ARTICLE; CELL CULTURE; CELL NUCLEUS; CODON; ENDOMETRIUM TUMOR; FEMALE; GENETICS; HUMAN; METABOLISM; MISSENSE MUTATION; NUCLEOTIDE SEQUENCE; PATHOLOGY;

BETA CATENIN; CELL NUCLEUS; CODON; CYTOSKELETAL PROTEINS; DNA MUTATIONAL ANALYSIS; ENDOMETRIAL NEOPLASMS; FEMALE; HUMANS; MUTATION, MISSENSE; TRANS-ACTIVATORS; TUMOR CELLS, CULTURED;

EID: 0034146020     PISSN: 1021335X     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (1)

1. Hulsken J, Birchmeier W and Behrens J: E-cadherin and APC compete for the interaction with β-catenin and cytoskelton. J Cell Biol 127: 2061-2069, 1994.