Cell death in the central nervous system: Therapeutic possibilities?

Regional Anesthesia and Pain Medicine

Volumn 25, Issue 1, 2000, Pages 22-25

  Giffard, Rona G ^a  

^a STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMPA RECEPTOR; CASPASE; CYTOCHROME C OXIDASE; DEXTROMETHORPHAN; DIZOCILPINE; GLUTAMATE RECEPTOR ANTAGONIST; GLYCERYL TRINITRATE; KAINIC ACID RECEPTOR; KETAMINE; MEMANTINE; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; NEUROPROTECTIVE AGENT; PHENCYCLIDINE; PROTEIN BAX; PROTEIN BCL 2; RILUZOLE;

APOPTOSIS; ARTICLE; BRAIN PROTECTION; CELL DEATH; CENTRAL NERVOUS SYSTEM; HUMAN; NERVE CELL NECROSIS; NEUROMODULATION; NEUROPROTECTION; NEUROTOXICITY; NONHUMAN; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

EID: 0033958612     PISSN: 10987339     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1098-7339(00)80006-1     Document Type: Article

References (4)

1. Lipton SA, Rosenberg PA. Mechanisms of disease: Excitatory amino acids as a final common pathway for neurologic disorders. NEnglJMed 1994;330:613-622.
2. Schultz JB, Weller M, Moskowitz MA. Caspases as treatment targets in stroke and neurodegenerative diseases. Ann Neural 1999:45:421-429.
3. Apoptosis, special section Science 1998:281:1301-1326. This includes reviews on caspases, bcl-2 family members, mitochondria in apoptosis, and death receptor signalling.
4. Fiskum G, Murphy AN, Beal MR Mitochondria in neurodegeneration: Acute ischemia and chronic neurodegenerative diseases. J Cereb Blood Flow Metab 1999;19:351369.