Molecular analysis of granulocytic sarcoma: A single center experience
Martinelli G, Ottaviani E, Testoni N, Visani G, Pagliani G, Tura S. Molecular analysis of granulocytic sarcoma: a single center experience. Haematologica 1999; 84: 380-2.
Molecular pathogenesis of the chromosome 16 inversion in the M4Eo subtype of acute myeloid leukemia
Liu P, Hajra A, Wijmenga C, et al. Molecular pathogenesis of the chromosome 16 inversion in the M4Eo subtype of acute myeloid leukemia. Blood 1995; 85: 2289-302.
Autologous peripheral blood stem cell transplantation in acute myeloblastic leukaemia and myelodysplastic syndrome patients: Evaluation of tumour cell contamination of leukaphereses by cytogenetic and molecular methods
Testoni N, Lemoli RM, Martinelli G, et al. Autologous peripheral blood stem cell transplantation in acute myeloblastic leukaemia and myelodysplastic syndrome patients: evaluation of tumour cell contamination of leukaphereses by cytogenetic and molecular methods Bone Marrow Transplant 1998; 22:1065-70.
Defining the absence of the CBFβ/MYH11 fusion transcript in patients with acute myeloid leukemia and inversion of chromosome 16 to predict long-term complete remission: A call for definitions
Marcucci G, Caligiuri MA, Bloomfield CD. Defining the absence of the CBFβ/MYH11 fusion transcript in patients with acute myeloid leukemia and inversion of chromosome 16 to predict long-term complete remission: a call for definitions. Blood 1997; 90:5022-4.
Prognosis value of residual disease monitoring by polymerase chain reaction in patients with CBFβ/MYH11-positive acute myeloblastic leukemia
Costello R, Sainty D, Blaise D, et al. Prognosis value of residual disease monitoring by polymerase chain reaction in patients with CBFβ/MYH11-positive acute myeloblastic leukemia. Blood 1997; 89:2222-3.
Detection and quantitation of the CBFβ/MYH11 transcripts associated with the inv(16) in presentation and follow-up samples from patients with AML
Evans PA, Short MA, Jack AS, et al. Detection and quantitation of the CBFβ/MYH11 transcripts associated with the inv(16) in presentation and follow-up samples from patients with AML. Leukemia 1997; 11: 364-9.
Competitive CBFβ/MYH11 reverse-transcriptase polymerase chain reaction for quantitative assessment of minimal residual disease during postremission therapy in acute myeloid leukemia with inversion(16): A pilot study
Laczika K, Novak M, Hilgarth B, et al. Competitive CBFβ/MYH11 reverse-transcriptase polymerase chain reaction for quantitative assessment of minimal residual disease during postremission therapy in acute myeloid leukemia with inversion(16): a pilot study. J Clin Oncol 1998; 16:1519-25.
Detection of minimal residual disease in patients with AML1/ETO-associated acute myeloid leukemia using a novel quantitative reverse transcription polymerase chain reaction assay
Marcucci G, Livak KJ, Bi W, et al. Detection of minimal residual disease in patients with AML1/ETO-associated acute myeloid leukemia using a novel quantitative reverse transcription polymerase chain reaction assay. Leukemia 1998; 12:1482-9.
The use of quantitative RT-PCR for bcr-abl in the clinical management of chronic myeloid leukemia
Martinelli G, Saglio G, Baccarani M, et al. The use of quantitative RT-PCR for bcr-abl in the clinical management of chronic myeloid leukemia. Haematologica 1998; 83(e-suppl. 12):18-25.
(1998)Haematologica, vol.83, Issue.12 E-SUPPL, pp. 18-25