The common deletion 657del5 in the Nibrin gene is not a major risk factor for B or T cell non-Hodgkin lymphoma in a pediatric population [2]

Leukemia

Volumn 14, Issue 8, 2000, Pages 1528-1529

  Rischewski, J ^a   Bismarck, P V ^a   Kabisch, H ^a   Janka Schaub, G ^a   Obser, T ^a   Schneppenheim, R ^a  

^a UNIVERSITY MEDICAL CENTER HAMBURG EPPENDORF   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

DNA; NIBRIN; POLYDEOXYRIBONUCLEOTIDE SYNTHASE; UNCLASSIFIED DRUG;

B CELL LYMPHOMA; CANCER RISK; CHILD; CHROMOSOME ABERRATION; CHROMOSOME ANALYSIS; CLINICAL FEATURE; CONTROLLED STUDY; FEMALE; GENE DELETION; GENE MUTATION; GENE SEQUENCE; GERMANY; HETEROZYGOSITY; HUMAN; IMMUNE DEFICIENCY; LETTER; MALE; NIJMEGEN BREAKAGE SYNDROME; NONHODGKIN LYMPHOMA; PEDIATRICS; PRIORITY JOURNAL; RISK ASSESSMENT; T CELL LYMPHOMA;

EID: 0033902941     PISSN: 08876924     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.leu.2401836     Document Type: Letter

References (5)

1. Nuclease activities in a complex of human recombination and DNA repair factors Rad50, Mre11, and p95
2. Nijmegen breakage syndrome
3. Nibrin, a novel DNA double-strand break repair protein, is mutated in Nijmegen breakage syndrome
4. Incidence of the major mutation for Nijmegen breakage syndrom in Czech Republic and Poland
5. Determination of the frequency of the common 657Del5 Nijmegen breakage syndrome mutation in the German population: No association with risk of breast cancer