Coffee, K-ras mutations and pancreatic cancer: A heterogeneous aetiology or an artefact?

Journal of Epidemiology and Community Health

Volumn 54, Issue 9, 2000, Pages 654-655

  Jacobsen, B K ^a   Heuch, I ^b  

^a UNIVERSITY OF TROMSØ   (Norway)

^b UNIVERSITY OF BERGEN   (Norway)

Author keywords

[No Author keywords available]

Indexed keywords

ARTICLE; ARTIFACT; CANCER EPIDEMIOLOGY; CANCER RISK; CARCINOGENESIS; COFFEE; CONTROLLED STUDY; DNA REPAIR; DRINKING; GENE MUTATION; HUMAN; MATHEMATICAL MODEL; METABOLISM; ONCOGENE K RAS; PANCREAS CANCER; PREVALENCE; RISK FACTOR; SMOKING;

CAUSALITY; COFFEE; EPIDEMIOLOGY, MOLECULAR; GENES, RAS; GENETIC HETEROGENEITY; HUMANS; MUTATION; PANCREATIC NEOPLASMS; RISK; RISK FACTORS;

EID: 0033884777     PISSN: 0143005X     EISSN: None     Source Type: Journal    
DOI: 10.1136/jech.54.9.654     Document Type: Article

References (10)

1. Coffee, pancreatic cancer and the question of causation
2. Association between coffee drinking and K-ras mutations in exocrine pancreatic cancer
3. Coffee drinking, mortality, and cancer incidence: Results from a Norwegian prospective study
4. Use of alcohol, tobacco and coffee, and risk of pancreatic cancer
5. Coffee and cancer: A prospective study of 43,000 Norwegian men and women
6. Ras mutations and a cup of coffee: Cause, confounder, effect modifier, or what else?
7. Human cancer, carcinogenic exposures and mutation spectra
8. Statistical analysis of molecular epidemiology studies employing case-series
9. Mutational activation of K-ras in non-neoplastic exocrine pancreatic lesions in relation to cigarette smoking status