Absence of GAP-43 can protect neurons from death

Molecular and Cellular Neurosciences

Volumn 16, Issue 1, 2000, Pages 27-33

  Gagliardini, V ^a   Dusart, I ^b   Fankhauser, C ^a  

^a University of Zurich Brain Research Institute   (Switzerland)

^b PITIÉ SALPÊTRIÈRE HOSPITAL   (France)

Author keywords

[No Author keywords available]

Indexed keywords

NERVE GROWTH FACTOR; NEUROMODULIN; SEMAPHORIN 3A;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL PROTECTION; CELL SURVIVAL; CONTROLLED STUDY; GROWTH CONE; MOUSE; NERVE CELL NECROSIS; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PURKINJE CELL; SENSORY NERVE CELL;

ANIMALIA;

EID: 0033860076     PISSN: 10447431     EISSN: None     Source Type: Journal    
DOI: 10.1006/mcne.2000.0850     Document Type: Article

References (29)

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14. Neuropilin is a receptor for the axonal chemorepellent Semaphorin III
15. Hypoxia/ischemia induces dephosphorylation in rat brain neuromodulin/GAP-43 in vivo
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20. Dephosphorylation of neuromodulin by calcineurin
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29. GAP-43 amino terminal peptides modulate growth cone morphology and neurite outgrowth