c-Fos protein in photoreceptor cell death after photic injury in rats

Investigative Ophthalmology and Visual Science

Volumn 41, Issue 9, 2000, Pages 2755-2758

  Poon, H K ^a   Tso, M O M ^a   Lam, T T ^a  

^a HONG KONG EYE HOSPITAL   (Hong Kong)

Author keywords

[No Author keywords available]

Indexed keywords

DNA; PROTEIN C FOS;

ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL DEATH; CONTROLLED STUDY; IMMUNOHISTOCHEMISTRY; LIGHT; NONHUMAN; PHOTORECEPTOR CELL; PRIORITY JOURNAL; RAT; RETINA INJURY;

ANIMALS; APOPTOSIS; DNA; IMMUNOENZYME TECHNIQUES; IN SITU HYBRIDIZATION; IN SITU NICK-END LABELING; LIGHT; PHOTORECEPTORS, VERTEBRATE; PROTO-ONCOGENE PROTEINS C-FOS; RADIATION INJURIES, EXPERIMENTAL; RATS; RATS, INBRED LEW; RETINAL DISEASES; TIME FACTORS;

EID: 0033856328     PISSN: 01460404     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (12)

1. Photic injury triggers apoptosis of photoreceptor cells
2. The regulation and function of c-fos and other immediate early genes in the nervous system
3. The Fos complex and Fos-related antigens recognize sequence elements that contain AP-1 sites
4. Continuous c-fos expression precedes programmed cell death in vivo
5. The absence of c-fos prevents light-induced apoptotic cell death of photoreceptors in retinal degeneration in vivo
6. Retinal degeneration in the rd mouse in the absence of c-fos
7. The role of ocular pigmentation in protecting against retinal light damage
8. Apoptosis leads to photoreceptor degeneration in inherited retinal dystrophy of RCS rats
9. The c-Fos immunoreactivities in the developing and adult rat cerebella
10. c-Fos is not essential for apoptosis
11. Aberrant expression of c-Fos accompanies photoreceptor cell death in the rd mouse
12. Altered gene expression in neurons during programmed cell death: Identification of c-jun as necessary for neuronal apoptosis