Oxidative mechanisms in the toxicity of chromium and cadmium ions

Journal of Environmental Pathology, Toxicology and Oncology

Volumn 19, Issue 3, 2000, Pages 201-213

  Stohs, S J ^a   Bagchi, D ^a   Hassoun, E ^a   Bagchi, M ^a  

^a CREIGHTON UNIVERSITY SCHOOL OF PHARMACY AND HEALTH PROFESSIONS   (United States)

Author keywords

Apoptosis; Cadmium; Chromium; DNA damage; Free radicals; Glutathione depletion; Laser scanning confocal microscopy; Lipid peroxidation; Oxidative stress; P53 tumor suppressor gene; Redox cycling

Indexed keywords

CADMIUM; CHROMIUM; FREE RADICAL; GLUTATHIONE PEROXIDASE; PROTEIN KINASE C; REACTIVE OXYGEN METABOLITE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CHROMOSOME BREAKAGE; CONFOCAL LASER MICROSCOPY; CONTROLLED STUDY; DNA DAMAGE; FEMALE; LIPID PEROXIDATION; LIVER MICROSOME; NONHUMAN; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; TOXICITY; TUMOR SUPPRESSOR GENE;

ACETALDEHYDE; ACETONE; ANIMALS; CADMIUM; CADMIUM CHLORIDE; CATIONS; CELL SURVIVAL; CHROMATES; CHROMIUM; DNA DAMAGE; DOSE-RESPONSE RELATIONSHIP, DRUG; FEMALE; FORMALDEHYDE; GENES, P53; KINETICS; L-LACTATE DEHYDROGENASE; LETHAL DOSE 50; LIPID PEROXIDATION; MALONDIALDEHYDE; MICE; MICE, INBRED C57BL; MICROSOMES, LIVER; MITOCHONDRIA, LIVER; OXIDATIVE STRESS; PC12 CELLS; PROTEIN KINASE C; RATS; RATS, SPRAGUE-DAWLEY; REACTIVE OXYGEN SPECIES; TUMOR SUPPRESSOR PROTEIN P53;

EID: 0033850564     PISSN: 07318898     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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