Aspirin does not improve endothelial vasodilator function in Type 1 diabetes mellitus [7]

Diabetic Medicine

Volumn 17, Issue 7, 2000, Pages 556-557

  Skyrme Jones, R A P ^a   Meredith, I T ^a  

^a MONASH MEDICAL CENTRE   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

ACETYLCHOLINE; ACETYLSALICYLIC ACID; HEMOGLOBIN A1C; NITROPRUSSIDE SODIUM;

ADOLESCENT; ADULT; CLINICAL ARTICLE; CONTROLLED STUDY; ENDOTHELIUM; FOREARM BLOOD FLOW; HUMAN; INSULIN DEPENDENT DIABETES MELLITUS; LETTER; VASODILATATION;

ACETYLCHOLINE; ADULT; ASPIRIN; BLOOD GLUCOSE; BRACHIAL ARTERY; DIABETES MELLITUS, TYPE 1; ENDOTHELIUM, VASCULAR; FEMALE; FOREARM; HUMANS; MALE; NITROPRUSSIDE; PLETHYSMOGRAPHY; REFERENCE VALUES; REGIONAL BLOOD FLOW; VASODILATION;

EID: 0033847677     PISSN: 07423071     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1464-5491.2000.00311-7.x     Document Type: Letter

References (8)

1. Aspirin improves endothelial dysfunction in atherosclerosis
2. Aspirin protects endothelial cells from oxidative stress - Possible synergism with vitamin E
3. Aspirin increases ferritin synthesis in endothelial cells: A novel antioxidant pathway
4. Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C
5. Endothelial vasodilator function is related to LDL particle size and LDL vitamin E content in insulin-dependent diabetes mellitus
6. Continuous release of vasodilator prostanoids contributes to regulation of resting forearm blood flow in humans
7. Trial of repeated low-dose aspirin in diabetic angiopathy
8. Randomized trial of intravenous streptokinase, oral aspirin, both, or neither among 17187 cases of suspected acute myocardial infarction: ISIS-2