Effects of pinacidil on coronary Ca2+-myosin phosphorylation in cold potassium cardioplegia model

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 279, Issue 3 48-3, 2000, Pages

  Matsuda, Naruto ^a   Morgan, Kathleen G ^a,b   Sellke, Frank W ^a,c  

^a HARVARD MEDICAL SCHOOL   (United States)

^b BOSTON BIOMEDICAL RESEARCH INSTITUTE   (United States)

^c BETH ISRAEL DEACONESS MEDICAL CENTER   (United States)

Author keywords

Coronary vasoconstriction; Potassium channel; Vascular smooth muscle

Indexed keywords

ADENOSINE TRIPHOSPHATE; CALCIUM ION; GLIBENCLAMIDE; MYOSIN; MYOSIN LIGHT CHAIN; N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE; NITRIC OXIDE SYNTHASE INHIBITOR; PINACIDIL; POTASSIUM; POTASSIUM CHANNEL BLOCKING AGENT;

ANIMAL CELL; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM CELL LEVEL; CARDIOPLEGIA; CONTROLLED STUDY; CORONARY ARTERY; DOSE RESPONSE; DRUG MECHANISM; MALE; NONHUMAN; POTASSIUM TRANSPORT; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; RAT; REGULATORY MECHANISM; SMOOTH MUSCLE FIBER; VASOCONSTRICTION; VASOMOTOR REFLEX;

EID: 0033835842     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.2000.279.3.h882     Document Type: Article

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