Acquired slow-channel syndrome

Muscle and Nerve

Volumn 23, Issue 10, 2000, Pages 1582-1585

  Scola, R H ^a   Werneck, L C ^a   Iwamoto, F M ^a   Comerlato, E A ^a   Kay, C K ^a  

^a FEDERAL UNIVERSITY OF PARANÁ   (Brazil)

Author keywords

Electromyography; Myasthenia gravis; Neuromuscular junction; Slow channel syndrome

Indexed keywords

CHOLINERGIC RECEPTOR ANTIBODY; PYRIDOSTIGMINE;

ADULT; ARTICLE; CASE REPORT; CONGENITAL MYASTHENIC SYNDROME; GENETIC DISORDER; HUMAN; MALE; MUSCLE ACTION POTENTIAL; MUSCLE WEAKNESS; NERVE STIMULATION; NEUROMUSCULAR DISEASE; PRIORITY JOURNAL;

EID: 0033814801     PISSN: 0148639X     EISSN: None     Source Type: Journal    
DOI: 10.1002/1097-4598(200010)23:10<1582::AID-MUS16>3.0.CO;2-H     Document Type: Article

References (9)

1. Congenital myasthenic syndromes
2. A newly recognized congenital myasthenic syndrome attributed to a prolonged open time of the acetylcholine-induced ion channel
3. A β-subunit mutation in the acetylcholine receptor channel gate causes severe slow-channel syndrome
4. Congenital myasthenic syndrome caused by prolonged acetylcholine receptor channel openings due to a mutation in the M2 domain of the epsilon subunit
5. The slow-channel syndrome. Two new cases
6. Mutation of the acetylcholine receptor α subunit causes a slow-channel myasthenic syndrome by enhancing agonist binding affinity
7. Repetitive CMAPs: Mechanisms of neural and synaptic genesis
8. Acetylcholine receptor antibody as a diagnostic test for myasthenia gravis: Results in 153 validated cases and 2967 diagnostic assays
9. Acquired slowchannel syndrome: A form of myasthenia gravis with prolonged open time of the acetylcholine receptor channel