Dietary chloride does not correlate with urinary thromboxane in deoxycorticosterone acetate-treated rats

Journal of Laboratory and Clinical Medicine

Volumn 135, Issue 6, 2000, Pages 493-497

  Theriot, Judy A ^a   Passmore, John C ^a   Jimenez, Agnes E ^a   Fleming, John T ^a  

^a University of Louisville School of Medicine   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

EID: 0033806363     PISSN: 00222143     EISSN: None     Source Type: Journal    
DOI: 10.1067/mlc.2000.106808     Document Type: Article

References (14)

1. Separate hemodynamic roles for chloride and sodium in deoxycorticosterone acetate-salt hypertension
2. Modulating role for thromboxane in the tubuloglomerular feedback response in the rat
3. Concentration-dependent sodium chloride transport as the signal in feedback control of glomerular filtration rate
4. Influence of eicosanoids on renal function of DOCA-salt hypertensive rats
5. Selective preglomerular constriction to nerve stimulation in rat hydronephrotic kidneys
6. Facts and fallacies about measuring blood pressure in rats
7. Importance of chloride for deoxycorticosterone acetate-salt hypertension in the rat
8. Chloride alters renal blood flow autoregulation in deoxycorticosterone treated rats
9. Analysis of prostacyclin and thromboxane biosynteesis in cardiovascular disease
10. Thromboxane in cardiovascular disease
11. Role of thromboxane and prostacyclin release on photodynamic therapy-induced tumor destruction
12. Some statistical methods useful in circulation research
13. Enhanced release of endothelium-derived relaxing factor in mineralocorticoid hypertension
14. Feedback control of glomerular filtration rate: Site of the effector mechanism