Effects of oxidative stress on the expression of antioxidative defense enzymes in spontaneously hypertensive rat hearts

Free Radical Biology and Medicine

Volumn 29, Issue 7, 2000, Pages 612-619

  Csonka, Csaba ^a   Pataki, Tunde ^b   Kovacs, Peter ^b   Müller, Sebastian L ^a   Schroeter, Matthias L ^a   Tosaki, Arpad ^b   Blasig, Ingolf E ^a,c  

^a LEIBNIZ INSTITUT FÜR MOLEKULARE PHARMAKOLOGIE   (Germany)

^b Institute of Medicine   (Hungary)

^c NONE   (Germany)

Author keywords

Catalase; Free radicals; Gene expression; Myocardial hypertrophy; Oxidative stress; Rat heart; Superoxide dismutase

Indexed keywords

ANTIOXIDANT; CATALASE; FREE RADICAL; SUPEROXIDE DISMUTASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; GENE EXPRESSION; HEART DILATATION; HEART VENTRICLE HYPERTROPHY; HYPERTENSION; MALE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; SPONTANEOUSLY HYPERTENSIVE RAT; STRESS;

ANIMAL; CATALASE; COMPARATIVE STUDY; GENE EXPRESSION REGULATION, ENZYMOLOGIC; GLUTATHIONE PEROXIDASE; HYDROGEN PEROXIDE; HYPERTENSION; MALE; MYOCARDIUM; OXIDATIVE STRESS; RATS; RATS, INBRED SHR; RATS, INBRED WKY; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; RNA, MESSENGER; SUPEROXIDE DISMUTASE; SUPPORT, NON-U.S. GOV'T; TRANSCRIPTION, GENETIC;

EID: 0033805227     PISSN: 08915849     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0891-5849(00)00365-8     Document Type: Article

References (5)

1. 4-Hydroxynonenal, a novel indicator of lipid peroxidation for reperfusion injury of the myocardium
2. The spontaneously hypertensive rat as a model of the transition from compensated left ventricular hypertrophy to failure
3. Increased cardiomyocyte apoptosis during the transition to heart failure in the spontaneously hypertensive rat
4. 1-adrenoceptor