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Volumn 12, Issue 9, 2000, Pages 3331-3342
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The extracellular matrix molecule tenascin-R and its HNK-1 carbohydrate modulate perisomatic inhibition and long-term potentiation in the CA1 region of the hippocampus
a a a a a a |
Author keywords
GABA(A) receptors; HNK 1 carbohydrate; LTP; Perisomatic inhibition; Synaptic transmission; Tenascin R
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Indexed keywords
4 AMINOBUTYRIC ACID;
4 AMINOBUTYRIC ACID A RECEPTOR;
CARBOHYDRATE DERIVATIVE;
MONOCLONAL ANTIBODY;
NERVE CELL ADHESION MOLECULE;
TENASCIN;
4 AMINOBUTYRIC ACID RELEASE;
ANIMAL TISSUE;
ARTICLE;
CELLULAR DISTRIBUTION;
CONTROLLED STUDY;
EXTRACELLULAR MATRIX;
HIPPOCAMPUS;
IMMUNOCYTOCHEMISTRY;
LONG TERM POTENTIATION;
MOUSE;
NERVE CELL PLASTICITY;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
SYNAPTIC INHIBITION;
SYNAPTOSOME;
ANIMAL;
ANTIBODIES, MONOCLONAL;
ANTIGENS, CD57;
EXCITATORY POSTSYNAPTIC POTENTIALS;
EXTRACELLULAR MATRIX PROTEINS;
HIPPOCAMPUS;
LONG-TERM POTENTIATION;
MEMBRANE POTENTIALS;
MICE;
MICE, INBRED C57BL;
MICE, MUTANT STRAINS;
NEURAL CELL ADHESION MOLECULES;
NEURAL INHIBITION;
NEUTRALIZATION TESTS;
PARVALBUMINS;
PATCH-CLAMP TECHNIQUES;
PYRAMIDAL CELLS;
RECEPTORS, GABA-A;
SUPPORT, NON-U.S. GOV'T;
SYNAPTIC TRANSMISSION;
TENASCIN;
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EID: 0033798562
PISSN: 0953816X
EISSN: None
Source Type: Journal
DOI: 10.1046/j.1460-9568.2000.00216.x Document Type: Article |
Times cited : (106)
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References (51)
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