From coal mine dust to quartz: Mechanisms of pulmonary pathogenicity

Inhalation Toxicology

Volumn 12, Issue SUPPL. 3, 2000, Pages 7-14

  Castranova, V ^a  

^a NATIONAL INSTITUTE FOR OCCUPATIONAL SAFETY AND HEALTH   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

EID: 0033779044     PISSN: 08958378     EISSN: None     Source Type: Journal    
DOI: 10.1080/08958370050164842     Document Type: Conference Paper

References (26)

1. Patterns of secretion of transforming growth factor-alpha in experimental silicosis. Acute and subacute effects of cristobalite exposure in the rat
2. Silica-induced chemokine expression in alveolar type II cells is mediated by TNF-alpha
3. Antioxidant treatment attenuates cytokine and chemokine levels in murine macrophages following silica exposure
4. Comparison of inducible nitric oxide synthase gene expression and lung inflammation following intratracheal instillation of silica, coal, carbonyl iron, or titanium dioxide in rats
5. Particulates and the airways: Basic biological mechanisms of pulmonary pathogenicity
6. Coal dust
7. Silicosis and coal workers' pneumoconiosis
8. Role of surface free radicals in the pathogenicity of silica
9. Role of hydroxyl radical in silica-induced NFκB activation in macrophages
10. Coal: Response of cultured mammalian cells corresponds to prevalence of coal workers' pneumoconiosis
11. Freshly fractured crystalline silica induces activator protein-1 activation through ERKs and p38 MAPK
12. Crystalline silica and silicosis
13. TNFα and increased chemokine expression in rat lung after particle exposure
14. Tumor necrosis factor-induced release of platelet-derived growth factor from endothelial cells
15. 2+ content: Possible role in coal workers' pneumoconiosis
16. Potential biomarkers of coal workers' pneumoconiosis
17. How silicosis and coal workers' pneumoconiosis develop - A cellular assessment
18. Tumor necrosis factor is chemotactic for monocytes and polymorphonuclear leukocytes
19. Physico-chemical properties of silica in relation to its toxicity
20. Quartz hemolysis as related to its surface functionalities
21. Requirement for tumor necrosis factor for development of silica-induced pulmonary fibrosis
22. Silica-induced pulmonary inflammation in rats: Activation of NFκB and its suppression by dexamethasone
23. Diesel exhaust particles induced release of interleukin 6 and 8 by (primed) human bronchial epithelial cells (BEAS 2B) in vitro
24. Lung epithelial cell (A549) interaction with unopsonized environmental particulates: Quantitation of particle-specific binding and IL-8 production
25. Generation of oxygen radicals by minerals and its correlation to cytotoxicity
26. Generation of free radicals from freshly fractured silica dust: Potential role in acute silica-induced lung injury