Activation of transcription factors by diesel exhaust particles in human bronchial epithelial cells in vitro

Inhalation Toxicology

Volumn 12, Issue SUPPL. 3, 2000, Pages 359-364

  Bonvallot, V ^a   Baeza Squiban, A ^a   Boland, S ^a   Marano, F ^a  

^a UNIVERSITÉ PARIS DESCARTES   (France)

Author keywords

[No Author keywords available]

Indexed keywords

EID: 0033771287     PISSN: 08958378     EISSN: None     Source Type: Journal    
DOI: 10.1080/08958378.2000.11463246     Document Type: Conference Paper

References (10)

1. Diesel exhaust particles increase NF-κB DNA binding activity and C-fos proto-oncogene expression in human bronchial epithelial cells
2. The effect of diesel exhaust particles on cell function and release of inflammatory mediators from human bronchial epithelial cells in vitro
3. Diesel exhaust particles are taken up by human airway epithelial cells in vitro and alter cytokine production
4. Mechanisms of GM-CSF increase by diesel exhaust particles in human airway epithelial cells
5. Diesel exhaust particles induce local IgE production in vivo and alter the pattern of IgE messenger RNA isoforms
6. 2 and antioxidants have opposite effects on activation of NF-κB and AP1 in intact cells: AP1 as secondary antioxidant responsive factor
7. Asbestos induction of nuclear transcription factors and interleukin 8 gene regulation
8. Diesel exhaust particles induced release of IL6 and 8 by (primed) human bronchial epithelial cells (BEAS 2B) in vitro
9. Diesel exhaust particles induce NF-κB activation in human bronchial epithelial cells in vitro: Importance in cytokine transcription
10. ETS1, NF-κB and AP-1 synergistically transactivate the human GM-CSF promoter