Ultrastructure and phagocytic activity of rat peritoneal macrophages exposed to low temperatures in vitro

Cryobiology

Volumn 41, Issue 1, 2000, Pages 66-71

  Salman, H ^a,b   Bergman, M ^a,b   Bessler, H ^a,b   Alexandrova, S ^a,b   Djaldetti, M ^a,b  

^a RABIN MEDICAL CENTER   (Israel)

^b TEL AVIV UNIVERSITY   (Israel)

Author keywords

Hypothermia; Macrophages; Phagocytosis; Ultrastructure

Indexed keywords

ANIMAL CELL; ANIMAL EXPERIMENT; ARTICLE; CELL KILLING; CELL ULTRASTRUCTURE; COLD STRESS; HYPOTHERMIA; LOW TEMPERATURE PROCEDURES; LYMPHOCYTE TRANSFORMATION; MACROPHAGE; NONHUMAN; PERITONEUM MACROPHAGE; PHAGOCYTOSIS; PRIORITY JOURNAL; RAT;

ANIMALIA; BACTERIA (MICROORGANISMS);

EID: 0033770404     PISSN: 00112240     EISSN: None     Source Type: Journal    
DOI: 10.1006/cryo.2000.2267     Document Type: Article

References (13)

1. The effect of stress in vivo on the function of mouse macrophages in vitro
2. The effects of hypothermia on neutrophil function in vitro
3. Substance P and stress-induced changes in macrophages
4. Immunosuppression in mice induced by cold water stress
5. Studies on ultrastructural changes in perivascular cells (F.G.P.) of small cerebral vessels in the initial stage after cold injury - Chronological observations on F.G.P. within 24 hours
6. Immune cell and cortisol responses to physically and pharmacologically induced lowering of body core temperature
7. Increased superoxide anion production by immunologically activated and chemically elicited macrophages
8. Ultrastructural changes during continuous retrograde warm and mild hypothermic blood cardioplegia for coronary bypass operations
9. Temperature sensitivity on proliferation and morphologic alteration of human esophageal carcinoma cells in culture
10. Susceptibility of human and porcine neutrophils to hypothermia in vitro
11. Cold shock injury in animal cells
12. Mild intraoperative hypothermia reduces production of reactive oxygen intermediates by polymorphonuclear leukocytes
13. Endogenous substance P mediates cold water stress-induced increase in interleukin-6 secretion from peritoneal macrophages