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Volumn 115, Issue 4, 2000, Pages 746-752

Glucocorticoids augment the chemically induced production and gene expression of interleukin-1α through NF-κB and AP-1 activation in murine epidermal cells

Author keywords

Contact hypersensitivity; Cytokine; Glucocorticoids; Hapten; Keratinocyte

Indexed keywords

CYTOKINE; GLUCOCORTICOID; HAPTEN; HYDROCORTISONE; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERLEUKIN 10; INTERLEUKIN 1ALPHA; IRRITANT AGENT; PYRROLIDINE DITHIOCARBAMATE; STAPHYLOCOCCUS ENTEROTOXIN B; TRANSCRIPTION FACTOR AP 1; TUMOR NECROSIS FACTOR ALPHA;

EID: 0033770033     PISSN: 0022202X     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1523-1747.2000.00101.x     Document Type: Article
Times cited : (15)

References (28)
  • 14
    • 0028824168 scopus 로고
    • Removal of the majority of epidermal Langerhans cells by topical or systemic steroid application enhances the effector phase of murine contact hypersensitivity
    • (1995) J Immunol , vol.155 , pp. 4207-4217
    • Grabbe, S.1    Luger, A.T.2
  • 16
    • 0022982306 scopus 로고
    • Hydrocortisone reduces both constitutive and UV-elicited release of epidermal thymocyte activating factor (ETAF) by cultured keratinocytes
    • (1986) J Invest Dermatol , vol.87 , pp. 570-573
    • Kupper, T.S.1    McGuire, J.2
  • 21
    • 0027056242 scopus 로고
    • Systemic suppression of delayed-type hypersensitivity by supernatants from UV-irradiated keratinocytes. An essential role for lerarinocyte-derived IL-10
    • (1992) J Immunol , vol.15 , pp. 3865-3871
    • Rivas, J.M.1    Ullrich, S.E.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.