Adenosine and ATP cause nitric oxide-dependent pulmonary vasodilation in fetal lambs

Biology of the Neonate

Volumn 78, Issue 3, 2000, Pages 220-229

  Konduri, G Ganesh ^a   Mital, Seema ^b  

^a MEDICAL COLLEGE OF WISCONSIN   (United States)

^b WAYNE STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Adenosine; ATP; Fetal lung; K+ channels; Nitric oxide

Indexed keywords

15 HYDROXY 11ALPHA,9ALPHA EPOXYMETHANOPROSTA 5,13 DIENOIC ACID; ADENOSINE; ADENOSINE TRIPHOSPHATASE (POTASSIUM); GLIBENCLAMIDE; N(G) NITROARGININE; N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE; NITRIC OXIDE SYNTHASE INHIBITOR; PINACIDIL; POTASSIUM CHANNEL; POTASSIUM CHANNEL BLOCKING AGENT; POTASSIUM CHANNEL STIMULATING AGENT; THROMBOXANE RECEPTOR STIMULATING AGENT;

ANIMAL EXPERIMENT; ARTERY DILATATION; ARTICLE; CONCENTRATION RESPONSE; CONTROLLED STUDY; DRUG EFFECT; FETUS; LAMB; LUNG ARTERY PRESSURE; LUNG BLOOD FLOW; LUNG PRESSURE; LUNG VASCULAR RESISTANCE; NONHUMAN; PRIORITY JOURNAL; PULMONARY ARTERY;

EID: 0033767480     PISSN: 00063126     EISSN: None     Source Type: Journal    
DOI: 10.1159/000014274     Document Type: Article

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