Endothelin-1-induced contraction of rat thoracic aorta depends on calcium entry through three types of calcium channel

Journal of Cardiovascular Pharmacology

Volumn 36, Issue 5 SUPPL. 1, 2000, Pages

  Zhang, X F ^a   Iwamuro, Y ^a   Okamoto, Y ^a   Kawanabe, Y ^a   Masaki, T ^a   Miwa, S ^a  

^a KYOTO UNIVERSITY FACULTY OF MEDICINE   (Japan)

Author keywords

Endothelin 1 (ET 1); Intracellular free Ca2+ concentration; LOE 908; SK and F 96365; Vasoconstriction; Voltage independent calcium channel

Indexed keywords

1 [2 (4 METHOXYPHENYL) 2 [3 (4 METHOXYPHENYL)PROPOXY]ETHYL] 1H IMIDAZOLE; CALCIUM CHANNEL; CALCIUM ION; CATION CHANNEL; ENDOTHELIN 1;

ANIMAL TISSUE; AORTA CONSTRICTION; ARTICLE; CALCIUM CELL LEVEL; CALCIUM TRANSPORT; NONHUMAN; PRIORITY JOURNAL; RAT; THORACIC AORTA;

EID: 0033761189     PISSN: 01602446     EISSN: None     Source Type: Journal    
DOI: 10.1097/00005344-200036001-00085     Document Type: Article

References (4)

1. 2+ entry pathway in endothelin-1-induced contraction and its suppression by nitric oxide
2. 2+ entry channels in endothelin-1-induced contraction of rat aorta
3. 2+-permeable nonselective cation channel by endothelin-1 in A7r5 cells
4. 2+ channel blocker LOE 908