Aggressive multiple modality therapy for varicella-associated purpura fulminans [8]

British Journal of Haematology

Volumn 110, Issue 4, 2000, Pages 1012-1013

  Busuttil, D P ^a   Hay, C R M ^a   Lewis, M A ^b   Wynn, R F ^b  

^a MANCHESTER ROYAL INFIRMARY   (United Kingdom)

^b ROYAL MANCHESTER CHILDREN S HOSPITAL   (United Kingdom)

Author keywords

Factor V Leiden; Heparin; Plasma exchange; Protein S; Varicella associated purpura fulminans

Indexed keywords

ANTITHROMBIN; BLOOD CLOTTING FACTOR 10A INHIBITOR; D DIMER; DALTEPARIN; FIBRINOGEN; FRESH FROZEN PLASMA; HEPARIN; METHYLPREDNISOLONE; PROTEIN C; PROTEIN S;

AUTOIMMUNE DISEASE; CASE REPORT; CHICKENPOX; CHILD; CLINICAL FEATURE; DIAGNOSTIC PROCEDURE; DISEASE COURSE; FULMINATING PURPURA; HUMAN; HYPOFIBRINOGENEMIA; IMMUNOSUPPRESSIVE TREATMENT; LETTER; MALE; PLASMA TRANSFUSION; PLASMAPHERESIS; PRIORITY JOURNAL; PROTEIN S DEFICIENCY; TREATMENT OUTCOME;

ANTICOAGULANTS; CHICKENPOX; CHILD; COMBINED MODALITY THERAPY; CRYOGLOBULINS; FACTOR V; HEPARIN; HERPESVIRUS 3, HUMAN; HUMANS; MALE; PLASMA EXCHANGE; PROTEIN S DEFICIENCY; PROTHROMBIN; PURPURA, SCHOENLEIN-HENOCH;

EID: 0033760393     PISSN: 00071048     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1365-2141.2000.02270-9.x     Document Type: Letter

References (2)

1. Postinfectious purpura fulminans caused by an autoantibody directed against Protein S
2. Varicella and thrombotic complications associated with transient Protein C and Protein S deficiencies in children