An immunohistochemical study on cathepsin D in human hippocampus

Histochemical Journal

Volumn 32, Issue 8, 2000, Pages 505-508

  Nogami, Makoto ^a   Takatsu, Akihiro ^b   Endo, Noriko ^a   Ishiyama, Ikuo ^a  

^a TEIKYO UNIVERSITY SCHOOL OF MEDICINE   (Japan)

^b JIKEI UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

CATHEPSIN D;

ARTICLE; BRAIN ISCHEMIA; CLINICAL ARTICLE; GLIA CELL; HIPPOCAMPUS; HUMAN; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY; IMMUNOREACTIVITY; NERVE CELL DEGENERATION; NEUROPATHOLOGY; PRIORITY JOURNAL;

ADOLESCENT; ADULT; AGED; AGED, 80 AND OVER; AUTOPSY; CATHEPSIN D; FEMALE; HIPPOCAMPUS; HUMANS; IMMUNOHISTOCHEMISTRY; INFANT; MALE; MIDDLE AGED;

ANIMALIA; HIPPOCAMPUS HIPPOCAMPUS;

EID: 0033756587     PISSN: 00182214     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1004108804132     Document Type: Article

References (9)

1. Increased expression of cathepsins E and D in reactive microglial cells associated with spongiform degeneration in the brain stem of senescence-accelerated mouse
2. The distribution of cathepsin D activity in adult and aging human brain regions
3. Alteration of protease levels in different brain areas of suicide victims
4. Cathepsins B and D in rat brain glia during experimentally induced neuropathological defects. An immunocytochemical approach
5. Lysosomal proteinases as putative diagnostic tools in human neuropathology: Alzheimer disease (AD) and schizophrenia
6. Cerebral hypoxia
7. Colocalization of lysosomal hydrolase and beta-amyloid in diffuse plaques of the cerebellum and striatum in Alzheimer's disease and Down's syndrome
8. A comparison of cathepsin B processing and distribution during neuronal death in rats following global ischemia or decapitation necrosis
9. Anoxia does not produce brain damage