Increased levels of soluble tumor necrosis factor receptors in patients with aplastic anemia and myelodysplastic syndrome [3]

American Journal of Hematology

Volumn 65, Issue 4, 2000, Pages 326-327

  Shinohara, Kenji ^a   Muraki, Kazuhiko ^a   Ota, Itsuro ^a   Nawata, Rhohei ^b   Oeda, Eiichi ^b   Takahashi, Toru ^c  

^a Yamaguchi Prefectual Central Hospital   (Japan)

^b Shimonoseki Kosei Hospital   (Japan)

^c Department of Surgery   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

FAS ANTIGEN; FAS LIGAND; TUMOR NECROSIS FACTOR ALPHA; TUMOR NECROSIS FACTOR RECEPTOR;

APLASTIC ANEMIA; APOPTOSIS; CLINICAL ARTICLE; CLINICAL FEATURE; CONTROLLED STUDY; DISEASE ASSOCIATION; HUMAN; LETTER; MYELODYSPLASTIC SYNDROME; PANCYTOPENIA; PRIORITY JOURNAL;

ANEMIA, APLASTIC; HUMANS; MYELODYSPLASTIC SYNDROMES; RECEPTORS, TUMOR NECROSIS FACTOR;

EID: 0033739941     PISSN: 03618609     EISSN: None     Source Type: Journal    
DOI: 10.1002/1096-8652(200012)65:4<326::AID-AJH17>3.0.CO;2-Q     Document Type: Letter

References (6)

1. + human marrow cells is induced by interferon γ and tumor necrosis factor α and potentiates cytokine-mediated hematopoietic suppression in vitro
2. Apoptosis by death factor
3. Apoptosis signaling by death receptors
4. Soluble receptors for tumor necrosis factor in clinical laboratory diagnosis
5. Ligand passing: The 75-kDa tumor necrosis factor (TNF) receptor recruits TNF signaling by the 55-kDa TNF receptor
6. Increased production of tumor necrosis factor-α by peripheral blood mononuclear cells in patients with aplastic anemia