Commentary: Potential neurobiologic mechanisms through which metabolic disorders could relate to autism

Journal of Autism and Developmental Disorders

Volumn 30, Issue 5, 2000, Pages 471-473

  Johnston, Michael V ^a  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AUTISM; HUMAN; METABOLIC DISORDER; NEUROBIOLOGY; NOTE; PRIORITY JOURNAL; RETT SYNDROME;

AUTISTIC DISORDER; BRAIN; CHILD; CHILD, PRESCHOOL; HUMANS; RETT SYNDROME;

EID: 0033737852     PISSN: 01623257     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1005516010454     Document Type: Note

References (10)

1. Rett syndrome is caused by mutations in X-linked MeCP2, encoding methy-CpG-binding protein 2
2. Neuroanatomic observations of the brain in autism
3. Development of amino acid receptors in frontal cortex from girls with Rett syndrome
4. Developmental changes in brain serotonin synthesis capacity in autistic and nonautistic children
5. Hypoplasia of cerebellar vernal lobules VI and VII in autism
6. Recent progress in the neurology of learning: Memory molecules in the developing brain
7. Rett syndrome: A disorder affecting early brain growth
8. Neuropathological findings in the Rubinstein-Tabyi syndrome
9. Neurobiology of autism
10. Hippocampus in autism: A Golgi analysis