High frequency allelic loss on chromosome 17p13.3-p11.1 in esophageal squamous cell carcinomas from a high incidence area in northern China

Carcinogenesis

Volumn 21, Issue 11, 2000, Pages 2019-2026

  Huang, Jing ^a   Hu, Nan ^b   Goldstein, Alisa M ^b   Emmert Buck, Michael R ^a   Tang, Ze Zhong ^b   Roth, Mark J ^b   Wang, Quan Hong ^b   Dawsey, Sanford M ^b   Han, Xiao You ^b   Ding, Ti ^b   Li, Guang ^b   Giffen, Carol ^c   Taylor, Philip R ^a  

^a NATIONAL CANCER INSTITUTE   (United States)

^b SHANXI CANCER HOSPITAL   (China)

^c INFORMATION MANAGEMENT SERVICES INC   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALDEHYDE DEHYDROGENASE; MICROSATELLITE DNA; PROTEIN P53; ZINC FINGER PROTEIN;

ADULT; AGED; ALLELE; ARTICLE; CANCER INCIDENCE; CANCER RISK; CARCINOGENESIS; CHINA; CHROMOSOME 17P; CHROMOSOME LOSS; CONTROLLED STUDY; DIGESTIVE SYSTEM CANCER; ESOPHAGUS CARCINOMA; FAMILY HISTORY; FEMALE; GENE DELETION; GENE FREQUENCY; GENE LOCUS; GENETIC ANALYSIS; GENETIC LINKAGE; HETEROZYGOSITY LOSS; HIGH RISK POPULATION; HUMAN; LIFESTYLE; MAJOR CLINICAL STUDY; MALE; MARKER GENE; PRIORITY JOURNAL; SQUAMOUS CELL CARCINOMA; TUMOR SUPPRESSOR GENE;

EID: 0033737598     PISSN: 01433334     EISSN: None     Source Type: Journal    
DOI: 10.1093/carcin/21.11.2019     Document Type: Article

References (29)

1. Recent progress in research on esophageal cancer in China
2. Epidemiology of esophageal cancer in China
3. A survey of the familial aggregation of esophageal cancer in Yangcheng county, Shanxi Province
4. Familial aggregation of esophageal cancer in the high-incidence areas of North China: A strategy of control through genetic analysis
5. Genetic factor in the etiology of esophageal cancer and the strategy of its prevention in high-incidence areas of North China
6. Familial aggregation of esophageal cancer in Yangcheng County, Shanxi Province, China
7. Family history of esophageal cancer in Shanxi Province, China
8. Amplification of the EGF receptor and c-myc genes in human esophageal cancer
9. Coamplification of the hst-1 and int-2 gene in human cancer
10. Amplification of epidermal growth factor receptor gene but no evidence of ras mutation in primary human esophageal cancers
11. Frequent mutation of the p53 gene in human esophageal carcinoma
12. Frequent loss of heterozygosity at the retinoblastoma locus in human esophageal cancer
13. Altered expression of the cyclin D1 and retinoblastoma genes in human esophageal cancer
14. Genetic alterations in esophageal cancer and their relevance to etiology and pathogenesis: A review
15. Allelotype study of esophageal carcinoma
16. Identification of novel regions of allelic loss from a genome-wide scan of esophageal squamous cell carcinoma in a high-risk Chinese population
17. Allelic loss in esophageal squamous cell carcinoma patients with and without family history of upper gastrointestinal tract cancer
18. The gene for human p53 cellular tumor antigene is located on chromosome 17 short arm (17p13)
19. Allelotype analysis of esophageal adenocarcinoma: Loss of heterozygosity occurs at multiple sites
20. Multiple target sites of allelic imbalance on chromosome 17 in Barrett's oesophageal cancer
21. Laser capture microdissection
22. Laser capture microdissection: Molecular analysis of tissue
23. Allelic deletion on chromosome 17p13.3 in early ovarian cancer
24. Identification of two candidate tumor suppressor genes on chromosome 17p13.3
25. P53 activates expression of HIC-1, a new candidate tumor suppressor gene on 17p13.3
26. Cancer-specific region of hypermethylation identified within the HIC-1 putative tumor suppressor gene in acute myeloid leukemia
27. Methylation of HIC-1 candidate tumor suppressor gene in human breast cancer