Keep in contact: The role of the contact system in infection and sepsis

Critical Care Medicine

Volumn 28, Issue 11, 2000, Pages 3765-3766

  Levi, M ^a  

^a UNIVERSITY OF AMSTERDAM   (Netherlands)

Author keywords

Blood coagulation; Blood pressure; Contact system; Kallikrein kinin system; Kinins; Sepsis; Streptococcus

Indexed keywords

BLOOD CLOTTING FACTOR 12; PREKALLIKREIN;

BLOOD CLOTTING; CRITICAL ILLNESS; DISEASE SEVERITY; EDITORIAL; IN VITRO STUDY; INFECTION; MOUSE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; SEPTIC SHOCK; THROMBOEMBOLISM; VEIN THROMBOSIS;

EID: 0033678610     PISSN: 00903493     EISSN: None     Source Type: Journal    
DOI: 10.1097/00003246-200011000-00049     Document Type: Editorial

References (14)

1. Biochemistry and physiology of blood coagulation
2. Contact system: A vascular biology modulator with anticoagulant, profibrinolytic, antiadhesive, and proinflammatory attributes
3. The demise of John Hageman
4. Williams trait: Human kininogen deficiency with diminished levels of plasminogen proactivator and prekallikrein associated with abnormalities of the Hageman factor-dependent pathways
5. Activation of the coagulation cascade in C1-inhibitor deficiencies
6. Disseminated intravascular coagulation
7. The contact system contributes to hypotension but not disseminated intravascular coagulation in lethal bacteremia: In vivo use of a monoclonal anti-factor XII antibody to block contact activation in baboons
8. Contact activation in shock caused by invasive group A Streptococcus pyogenes
9. Kallikrein-kinin system activation in streptococcal toxic shock syndrome
10. Quantification of plasma factor XIIa-C1-inhibitor and kallikrein-C1-inhibitor complexes in sepsis
11. Reduction of contact activation related fibrinolytic activity in factor XII deficient patients: Further evidence for the role of the contact system in fibrinolysis in vivo
12. Reevaluation of the incidence of thromboembolic complications in congenital factor XII deficiency: A study on 73 subjects from 14 Swiss families
13. Kinin activation in the blood of patients with sepsis
14. Alpha 2-macroglobulin-kallikrein complexes detect contact system activation in hereditary angioedema and human sepsis