Causes of hyper-CK-emia in a neurological department

Neurologist

Volumn 6, Issue 6, 2000, Pages 309-314

  Finsterer, J ^a   Matz, K ^a  

^a NONE   (Austria)

Author keywords

Asymptomatic CK elevation; Creatine phosphokinase; Epilepsy; Muscle enzymes; Myoglobinuria; Myopathy; Neuromuscular disorders; Stroke; Trauma

Indexed keywords

CREATINE KINASE; MUSCLE ENZYME;

ARTICLE; BRAIN INJURY; CREATINE KINASE BLOOD LEVEL; FEMALE; HUMAN; MAJOR CLINICAL STUDY; MALE; MYOPATHY; NEUROMUSCULAR DISEASE; PRIORITY JOURNAL; RETROSPECTIVE STUDY; SEIZURE; STROKE;

EID: 0033653532     PISSN: 10747931     EISSN: None     Source Type: Journal    
DOI: 10.1097/00127893-200006060-00001     Document Type: Article

References (13)

1. Asymptomatic hyper-CK-emia: An electrophysiologic and histopathologic study
2. Idiopathic hyper CKemia
3. Persistent hyperCKemia: Fourteen patients studied in retrospect
4. Pitfalls in interpreting serum creatine phosphokinase activity
5. Postictal serum creatine kinase in the diagnosis of seizure disorders
6. Factors affecting serum creatine phosphokinase levels in the general population: The role of race, activity and age
7. Increase of serum creatine-phosphokinase in catatonia: An investigation in 32 acute catatonic patients
8. Cerebrospinal fluid levels of myelin basic protein and creatine kinase BB as index of active demyelination
9. Persistent aseptic meningitis in Vogt-Koyanagi-Harada syndrome
10. Creatine kinase in human tissues
11. Elevated creatine phosphokinase levels associated with large muscle mass
12. Befunde bei 100 Patienten mit 'atiologisch ungeklarter' erhohung der serumkreatinkinaseaktivitat