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Volumn 96, Issue 26, 1999, Pages 15038-15043
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Human cells compromised for p53 function exhibit defective global and transcription-coupled nucleotide excision repair, whereas cells compromised for pRb function are defective only in global repair
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Author keywords
[No Author keywords available]
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Indexed keywords
ONCOPROTEIN;
PHOTOPROTEIN;
PROTEIN P53;
PYRIMIDINE DIMER;
RETINOBLASTOMA PROTEIN;
ARTICLE;
CANCER INHIBITION;
DNA DAMAGE;
DNA REPAIR;
EXCISION REPAIR;
GENOTOXICITY;
HUMAN;
HUMAN CELL;
LUNG FIBROBLAST;
MALIGNANT TRANSFORMATION;
PHOTOTOXICITY;
POLYMERASE CHAIN REACTION;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
SKIN FIBROBLAST;
TUMOR SUPPRESSOR GENE;
ULTRAVIOLET B RADIATION;
VIRUS CARCINOGENESIS;
WART VIRUS;
DNA REPAIR;
FIBROBLASTS;
HUMANS;
LI-FRAUMENI SYNDROME;
LUNG;
ONCOGENE PROTEINS, VIRAL;
PYRIMIDINE DIMERS;
RETINOBLASTOMA PROTEIN;
SKIN;
TRANSCRIPTION, GENETIC;
TUMOR SUPPRESSOR PROTEIN P53;
ULTRAVIOLET RAYS;
EUKARYOTA;
HUMAN PAPILLOMAVIRUS;
HUMAN PAPILLOMAVIRUS TYPES;
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EID: 0033592978
PISSN: 00278424
EISSN: None
Source Type: Journal
DOI: 10.1073/pnas.96.26.15038 Document Type: Article |
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Times cited : (85)
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References (61)
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