Estradiol repression of tumor necrosis factor-α transcription requires estrogen receptor activation function-2 and is enhanced by coactivators

Proceedings of the National Academy of Sciences of the United States of America

Volumn 96, Issue 26, 1999, Pages 15161-15166

  An, Jinping ^a   Ribeiro, Ralff C J ^b,c   Webb, Paul ^b   Gustafsson, Jan Åke ^d   Kushner, Peter J ^b   Baxter, John D ^b   Leitman, Dale C ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c UNIVERSITY OF BRASILIA   (Brazil)

^d KAROLINSKA INSTITUTE   (Sweden)

Author keywords

[No Author keywords available]

Indexed keywords

COMPLEMENTARY DNA; ESTRADIOL; ESTROGEN RECEPTOR; GLUCOCORTICOID RECEPTOR; MESSENGER RNA; REGULATOR PROTEIN; TUMOR NECROSIS FACTOR ALPHA;

ARTICLE; CONTROLLED STUDY; ESTROGEN ACTIVITY; GENE OVEREXPRESSION; GENE REPRESSION; GENETIC TRANSCRIPTION; HORMONAL REGULATION; HUMAN; HUMAN CELL; OSTEOLYSIS; OSTEOPOROSIS; POSTMENOPAUSE OSTEOPOROSIS; PRIORITY JOURNAL; PROMOTER REGION;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; BINDING SITES; ESTRADIOL; ESTROGEN RECEPTOR ALPHA; ESTROGEN RECEPTOR BETA; ESTROGEN RECEPTOR MODULATORS; GENE EXPRESSION REGULATION; LIGANDS; MUTATION; NUCLEAR PROTEINS; NUCLEAR RECEPTOR COACTIVATOR 2; RECEPTORS, ESTROGEN; RESPONSE ELEMENTS; SELECTIVE ESTROGEN RECEPTOR MODULATORS; SIMPLEXVIRUS; THYMIDINE KINASE; TRANSCRIPTION FACTOR AP-1; TRANSCRIPTION FACTORS; TRANSCRIPTION, GENETIC; TUMOR NECROSIS FACTOR-ALPHA;

HERPES; SIMPLEXVIRUS;

EID: 0033592892     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.96.26.15161     Document Type: Article

References (50)