Novel injury mechanism in anoxia and trauma of spinal cord white matter: glutamate release via reverse Na+-dependent glutamate transport.

The Journal of neuroscience : the official journal of the Society for Neuroscience

Volumn 19, Issue 14, 1999, Pages

  Li, S ^a   Mealing, G A ^a   Morley, P ^a   Stys, P K ^a  

^a OTTAWA HOSPITAL RESEARCH INSTITUTE   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

ABC TRANSPORTER; AMPA RECEPTOR; CARRIER PROTEIN; COTRANSPORTER; GLUTAMATE TRANSPORTER; GLUTAMIC ACID; SODIUM;

ACTION POTENTIAL; ANIMAL; ARTICLE; CELL HYPOXIA; DRUG ANTAGONISM; IMMUNOHISTOCHEMISTRY; IN VITRO STUDY; LONG EVANS RAT; MALE; MECHANICAL STRESS; METABOLISM; MYELIN SHEATH; NERVE FIBER; PATCH CLAMP; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; PRESSURE; RAT; SPINAL CORD; SPINAL CORD INJURY;

ACTION POTENTIALS; AMINO ACID TRANSPORT SYSTEM X-AG; ANIMALS; ATP-BINDING CASSETTE TRANSPORTERS; AXONS; CARRIER PROTEINS; CELL HYPOXIA; GLUTAMATE PLASMA MEMBRANE TRANSPORT PROTEINS; GLUTAMIC ACID; IMMUNOHISTOCHEMISTRY; MALE; MYELIN SHEATH; PATCH-CLAMP TECHNIQUES; PRESSURE; RATS; RATS, LONG-EVANS; RECEPTORS, AMPA; SODIUM; SPINAL CORD; SPINAL CORD INJURIES; STRESS, MECHANICAL; SYMPORTERS;

MLCS; MLOWN;

EID: 0033565016     PISSN: None     EISSN: 15292401     Source Type: Journal    
DOI: 10.1523/jneurosci.19-14-j0002.1999     Document Type: Article

References (0)