Hereditary ceruloplasmin deficiency increases advanced glycation end products in the brain

Neurology

Volumn 53, Issue 3, 1999, Pages 619-622

  Tajima, K ^a   Kawanami, T ^a   Nagai, R ^a   Horiuchi, S ^a   Kato, T ^a  

^a YAMAGATA UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

6 N CARBOXYMETHYLLYSINE; ADVANCED GLYCATION END PRODUCT; CERULOPLASMIN; FERROUS ION; HYDROGEN PEROXIDE; LYSINE; LYSINE DERIVATIVE; UNCLASSIFIED DRUG;

ADULT; ARTICLE; BASAL GANGLION; BRAIN PROTECTION; CASE REPORT; DEMENTIA; ENZYME DEFICIENCY; GENE MUTATION; HUMAN; HUMAN TISSUE; IRON METABOLISM; MALE; OXIDATIVE STRESS; PRIORITY JOURNAL; RETINA DEGENERATION;

EID: 0033546644     PISSN: 00283878     EISSN: None     Source Type: Journal    
DOI: 10.1212/wnl.53.3.619     Document Type: Article

References (10)