Control of separate pathogenic autoantibody responses marks MHC gene contributions to murine lupus

Proceedings of the National Academy of Sciences of the United States of America

Volumn 96, Issue 14, 1999, Pages 8098-8103

  Vyse, T J ^a   Halterman, R K ^a   Rozzo, S J ^a   Izui, S ^a   Kotzin, B L ^a  

^a UNIVERSITY OF COLORADO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANTINUCLEAR ANTIBODY; AUTOANTIBODY; DOUBLE STRANDED DNA; HISTONE; IMMUNOGLOBULIN G ANTIBODY; SINGLE STRANDED DNA;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANTIBODY BLOOD LEVEL; ANTIBODY PRODUCTION; ARTICLE; CHROMATIN; CHROMOSOME 16; CHROMOSOME 7; DISEASE PREDISPOSITION; FEMALE; GENE LOCATION; GENE LOCUS; GENE MAPPING; GENETIC HETEROGENEITY; GENETIC RISK; HAPLOTYPE; LUPUS ERYTHEMATOSUS NEPHRITIS; MAJOR HISTOCOMPATIBILITY COMPLEX; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL;

ANIMALS; ANTIBODY FORMATION; AUTOANTIBODIES; CHROMOSOME MAPPING; CROSSES, GENETIC; FEMALE; GENES, DOMINANT; GENOME; H-2 ANTIGENS; HAPLOTYPES; LUPUS ERYTHEMATOSUS, SYSTEMIC; LUPUS NEPHRITIS; MAJOR HISTOCOMPATIBILITY COMPLEX; MALE; MICE; MICE, INBRED STRAINS; POLYMORPHISM, RESTRICTION FRAGMENT LENGTH; RISK FACTORS;

EID: 0033529193     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.96.14.8098     Document Type: Article

References (37)