Mitochondrial antioxidant function is a potential mechanism for organ differences in interleukin-1-mediated tolerance to oxidative injury

American Journal of the Medical Sciences

Volumn 318, Issue 5, 1999, Pages 308-315

  Guidot, David M ^a,b,c   Kitlowski, Andrew D ^a   Hybertson, Brooks M ^a   Repine, John E ^a  

^a UNIVERSITY OF COLORADO   (United States)

^b ATLANTA VA MEDICAL CENTER   (United States)

^c EMORY UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Antioxidant; Kidney; Lung; Mitochondria; Superoxide

Indexed keywords

ANTIOXIDANT; INTERLEUKIN 1ALPHA; SUPEROXIDE;

ADULT RESPIRATORY DISTRESS SYNDROME; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; HEART MUSCLE ISCHEMIA; HYPEROXIA; LUNG INJURY; MALE; MITOCHONDRION; NONHUMAN; OXIDATIVE STRESS; RAT; REPERFUSION INJURY;

EID: 0033505255     PISSN: 00029629     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0002-9629(15)40644-5     Document Type: Article

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