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Volumn 15, Issue 6, 1999, Pages 1177-1183
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Use of novel plasmid constructs to demonstrate fludarabine triphosphate inhibition of nucleotide excision repair of a site-specific 1,2-d(GpG) intrastrand cisplatin adduct.
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Author keywords
[No Author keywords available]
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Indexed keywords
ADENOSINE TRIPHOSPHATE;
ANTINEOPLASTIC AGENT;
CISPLATIN;
CISPLATIN DNA ADDUCT;
CISPLATIN-DNA ADDUCT;
DNA;
DRUG DERIVATIVE;
FLUDARABINE;
OLIGONUCLEOTIDE;
RECOMBINANT DNA;
VIDARABINE;
ARTICLE;
BINDING SITE;
CELL CULTURE;
CHEMISTRY;
DNA ADDUCT;
DNA REPAIR;
DOSE RESPONSE;
DRUG EFFECT;
ENZYME SPECIFICITY;
GENETICS;
HUMAN;
METABOLISM;
MOLECULAR GENETICS;
NUCLEOTIDE SEQUENCE;
PLASMID;
SYNTHESIS;
ADENOSINE TRIPHOSPHATE;
ANTINEOPLASTIC AGENTS;
BASE SEQUENCE;
BINDING SITES;
CISPLATIN;
DNA;
DNA ADDUCTS;
DNA REPAIR;
DNA, RECOMBINANT;
DOSE-RESPONSE RELATIONSHIP, DRUG;
HUMANS;
MOLECULAR SEQUENCE DATA;
OLIGONUCLEOTIDES;
PLASMIDS;
SUBSTRATE SPECIFICITY;
TUMOR CELLS, CULTURED;
VIDARABINE;
MLCS;
MLOWN;
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EID: 0033426724
PISSN: 10196439
EISSN: None
Source Type: Journal
DOI: 10.3892/ijo.15.6.1177 Document Type: Article |
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Times cited : (10)
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References (0)
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