Ca2+-dependent permeability transition and complex I activity in lymphoblast mitochondria from normal individuals and patients with Huntington's or Alzheimer's disease

Annals of the New York Academy of Sciences

Volumn 893, Issue , 1999, Pages 365-368

  Panov, Alexander ^a   Obertone, Tracy ^a   Bennett Desmelik, Julie ^a   Greenamyre, J Timothy ^a  

^a EMORY UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE);

ALZHEIMER DISEASE; CONFERENCE PAPER; CONTROLLED STUDY; HUMAN; HUMAN CELL; HUNTINGTON CHOREA; LYMPHOBLAST; MEMBRANE PERMEABILITY; MITOCHONDRIAL MEMBRANE;

EID: 0033382256     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1111/j.1749-6632.1999.tb07856.x     Document Type: Conference Paper

References (6)

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3. 3. Nicholls, D.G. & S.L. Budd. 1998. Mitochondria and glutamate excitotoxicity. Biochim. Biophys. Acta 1366: 97-112.
4. 4. Zamzami, N. et al. 1996. Mitochondrial control of nuclear apoptosis. J. Exp. Med. 183: 1533-1544.
5. 5. Wallace, D.C. 1997. Mitochondrial DNA mutations and bioenergetic defects in aging and degenerative diseases. In The Molecular and Genetic Basis of Neurological Disease, 2d edit. R.N. Rosenberg et al., Eds. :237-269. Butterworth-Heinemann. Boston, Oxford, Johannesburg, Melbourne.
6. 6. Trounce, I.A. et al. 1996. Assessment of mitochondrial oxidative phosphorylation in patient muscle biopsies, lymphoblasts, and transmitochondrial cell lines. Methods Enzymol. 264: 484-509.