Mitochondrial 16189 variant, thinness at birth, and type-2 diabetes

Lancet

Volumn 353, Issue 9163, 1999, Pages 1499-1500

  Casteels, Kristina ^b   Ong, Ken ^b   Phillips, David ^b   Bendall, Holly ^b   Pembrey, Marcus ^b   Poulton, Jo ^b   Dunger, David ^a  

^a UNIVERSITY OF OXFORD   (United Kingdom)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

MITOCHONDRIAL DNA;

ARTICLE; GENOTYPE; HUMAN; INTRAUTERINE GROWTH RETARDATION; LOW BIRTH WEIGHT; MALE; NON INSULIN DEPENDENT DIABETES MELLITUS; PRIORITY JOURNAL;

EID: 0033135836     PISSN: 01406736     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0140-6736(98)05817-6     Document Type: Article

References (5)

1. Hales CN, Barker DJ, Clark PM, et al. Fetal and infant growth and impaired glucose tolerance at age 64. BMJ 1991; 303: 1019-22.
2. Ounsted M, Scott A, Ounsted C. Transmission through the female line of a mechanism constraining human fetal growth. Ann Hum Biol 1986; 13: 143-51.
3. Poulton J, Brown MS, Cooper A, Marchington DR, Phillips DI. A common mitochondrial DNA variant is associated with insulin resistance in adult life. Diabetologia 1998; 41: 54-58.
4. Dunger DB, Ong KK, Huxtable SJ, et al. Association of the INS VNTR with size at birth. Nat Genet 1998; 19: 98-100.
5. Ong KK, Phillips DI, Fall C, et al. The insulin gene VNTR, type 2 diabetes and birth weight. Nat Genet 1999; 21:262-63