Bcl-2 protects against beta-lapachone-mediated caspase 3 activation and apoptosis in human myeloid leukemia (HL-60) cells.

Oncology reports

Volumn 6, Issue 3, 1999, Pages 485-492

  Planchon, S M ^a   Wuerzberger Davis, S M ^a   Pink, J J ^a   Robertson, K A ^a   Bornmann, W G ^a   Boothman, D A ^a  

^a CASE WESTERN RESERVE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BETA LAPACHONE; BETA-LAPACHONE; CASP3 PROTEIN, HUMAN; CASPASE; CASPASE 3; NAPHTHOQUINONE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; PROTEIN BCL 2; PROTEINASE INHIBITOR;

APOPTOSIS; ARTICLE; BIOSYNTHESIS; CELL STRAIN HL 60; CYTOLOGY; DRUG EFFECT; ENZYME ACTIVATION; ENZYMOLOGY; GENE VECTOR; GENETICS; HUMAN; METABOLISM; PHYSIOLOGY;

APOPTOSIS; CASPASE 3; CASPASES; ENZYME ACTIVATION; GENETIC VECTORS; HL-60 CELLS; HUMANS; NAPHTHOQUINONES; POLY(ADP-RIBOSE) POLYMERASES; PROTEASE INHIBITORS; PROTO-ONCOGENE PROTEINS C-BCL-2;

EID: 0033129747     PISSN: 1021335X     EISSN: None     Source Type: Journal    
DOI: 10.3892/or.6.3.485     Document Type: Article

References (0)