The high cost of living

Trends in Genetics

Volumn 15, Issue 3, 1999, Pages 93-94

  Kunkel, Thomas A ^a  

^a NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

3 METHYLADENINE; ADENINE DERIVATIVE; GUANINE DERIVATIVE; HYPOXANTHINE; PYRIMIDINE DERIVATIVE; THYMINE; URACIL;

CANCER GENETICS; CONFERENCE PAPER; CYTOTOXICITY; DEAMINATION; DNA DAMAGE; DNA REPAIR; DNA STRAND BREAKAGE; EXCISION REPAIR; GENETIC SUSCEPTIBILITY; HUMAN; HYDROLYSIS; LIPID PEROXIDATION; METHYLATION; MUTAGENESIS; MUTATION; NUCLEOTIDE SEQUENCE; OXIDATION; PRIORITY JOURNAL; SPONTANEOUS MUTATION;

ANIMALS; DNA DAMAGE; DNA REPAIR; DNA REPLICATION; HUMANS; MUTATION; NEOPLASMS;

EID: 0033028426     PISSN: 01689525     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0168-9525(98)01664-3     Document Type: Conference Paper

References (6)

1. Lindahl T. Instability and decay of the primary structure of DNA. Nature. 362:1993;709-715.
2. Rink S.M.et al. Creation of RNA molecules that recognize the oxidative lesion 7,8-dihydro-8-hydroxy-2'-deoxyguanosine (8-oxodG) in DNA. Proc. Natl. Acad. Sci. U. S. A. 95:1998;11619-11624.
3. Wilson S.H. Mammalian base excision repair and DNA polymerase beta. Mutat. Res. 407:1998;203-215.
4. Kim K.et al. Involvement of flap endonuclease 1 in base excision DNA repair. J. Biol. Chem. 273:1998;8842-8848.
5. Holmquist G.P. Endogenous lesions, S-phase-independent spontaneous mutations, and evolutionary strategies for base excision repair. Mutat. Res. 400:1998;59-68.
6. Loeb L.A. Cancer cells exhibit a mutator phenotype. Adv. Cancer Res. 72:1998;25-56.