Truncated CBP protein leads to classical Rubinstein-Taybi syndrome phenotypes in mice: Implications for a dominant-negative mechanism

Human Molecular Genetics

Volumn 8, Issue 3, 1999, Pages 387-396

  Oike, Yuichi ^a   Hata, Akira ^b   Mamiya, Takayoshi ^c   Kaname, Tadashi ^a   Noda, Yukihiro ^c   Suzuki, Misao ^a   Yasue, Hirofumi ^a   Nabeshima, Toshitaka ^c   Araki, Kimi ^a   Yamamura, Ken Ichi ^a  

^a KUMAMOTO UNIV SCHOOL OF MEDICINE   (Japan)

^b HOKKAIDO UNIVERSITY   (Japan)

^c NAGOYA UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLIC AMP RESPONSIVE ELEMENT BINDING PROTEIN;

AMINO ACID SEQUENCE; AMNESIA; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BONE MATURATION; CLINICAL FEATURE; CONDITIONING; DEVELOPMENTAL DISORDER; FEAR; GENE INSERTION; GROWTH RETARDATION; HEART DISEASE; LONG TERM MEMORY; MAXILLA; MOUSE; NONHUMAN; PALATE; PHENOTYPE; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN DOMAIN; PROTEIN EXPRESSION; RUBINSTEIN SYNDROME; SHORT TERM MEMORY; SKELETON MALFORMATION;

ANIMALIA; MAMMALIA;

EID: 0033018277     PISSN: 09646906     EISSN: None     Source Type: Journal    
DOI: 10.1093/hmg/8.3.387     Document Type: Article

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