Expression of Ras-GRF in the SK-N-BE neuroblastoma accelerates retinoic-acid-induced neuronal differentiation and increases the functional expression of the IRK1 potassium channel

European Journal of Neuroscience

Volumn 11, Issue 3, 1999, Pages 959-966

  Tonini, Raffaella ^a   Mancinelli, Enzo ^a   Balestrini, Monica ^a   Mazzanti, Michele ^a   Martegani, Enzo ^a   Ferroni, Arnaldo ^a   Sturani, Emmapaola ^a   Zippel, Renata ^a  

^a UNIVERSITY OF MILAN   (Italy)

Author keywords

Activation of Ras; CDC25 Mm; Kir2.1; N myc; PD98059

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE INHIBITOR; POTASSIUM CHANNEL; POTASSIUM ION; RETINOIC ACID;

ARTICLE; HUMAN; HUMAN CELL; HYPERPOLARIZATION; NERVE CELL DIFFERENTIATION; NERVE CELL MEMBRANE POTENTIAL; NEUROBLASTOMA CELL; NORTHERN BLOTTING; PRIORITY JOURNAL;

EID: 0033008732     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1460-9568.1999.00504.x     Document Type: Article

References (2)

1. Arcangeli, A., Bianchi, L., Becchetti, A., Faravelli, L., Coronnello, M., Mini, E., Olivotto, M. & Wanke, E. (1995) A novel inward-rectifying K+ current with a cell-cycle dependence governs the resting potential of mammalian neuroblastoma cells. J. Plysiol., 489, 455-471.
2. Arcangeli, A., Rosati, B., Cherubim, A., Crociani, O., Fontana, L., Passani, B., Wanke, E. & Olivotto, M. (1998) Long term exposure to retinoic acid induces the expression of IRK1 channels in HERG channel-endowed neuroblastoma cells. Biochem. Biophys. Res. Commun., 244, 706-711.