Increased AP-1 DNA binding activity in PC12 cells treated with lead

Journal of Neurochemistry

Volumn 73, Issue 1, 1999, Pages 187-194

  Chakraborti, Tamal ^a   Kim, Kyung Ah ^a   Goldstein, Gary G ^a   Bressler, Joseph P ^a,b  

^a JOHNS HOPKINS UNIVERSITY   (United States)

^b KENNEDY KRIEGER INSTITUTE   (United States)

Author keywords

Activator protein 1 complex; Lead; PC12 cells; Protein kinase C

Indexed keywords

LEAD; PROTEIN KINASE C; PROTEIN KINASE C INHIBITOR; TRANSCRIPTION FACTOR AP 1;

ANIMAL CELL; ARTICLE; DENSITOMETRY; DNA BINDING; DOSE TIME EFFECT RELATION; ELECTROPHORETIC MOBILITY; ENZYME REGULATION; LEAD POISONING; NONHUMAN; PRIORITY JOURNAL; RAT; TRANSCRIPTION REGULATION;

ANIMALS; CEREBELLUM; DNA; DOSE-RESPONSE RELATIONSHIP, DRUG; GENES, FOS; GENES, JUN; KINETICS; ORGANOMETALLIC COMPOUNDS; PC12 CELLS; PHORBOL 12,13-DIBUTYRATE; PROTEIN BIOSYNTHESIS; PROTEIN KINASE C; PROTEIN SYNTHESIS INHIBITORS; RATS; TRANSCRIPTION FACTOR AP-1; TRANSFECTION;

ANIMALIA;

EID: 0033002940     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1471-4159.1999.0730187.x     Document Type: Article

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