Selective enhancement of production of IgE, IgG4, and Th2-cell cytokine during the rebound phenomenon in atopic dermatitis and prevention by suplatast tosilate

Annals of Allergy, Asthma and Immunology

Volumn 82, Issue 3, 1999, Pages 293-295

  Kimata, Hajime ^a  

^a Unitika Central Hospital   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

BUFEXAMAC; CYTOKINE; DEXAMETHASONE 17 VALERATE; IMMUNOGLOBULIN E; IMMUNOGLOBULIN G4; INTERLEUKIN 10; INTERLEUKIN 13; INTERLEUKIN 4; INTERLEUKIN 5; SUPLATAST TOSYLATE;

ADULT; ARTICLE; ATOPIC DERMATITIS; CLINICAL ARTICLE; CLINICAL TRIAL; CONTROLLED CLINICAL TRIAL; CONTROLLED STUDY; CYTOKINE PRODUCTION; DRUG EFFICACY; FEMALE; HUMAN; IMMUNOGLOBULIN PRODUCTION; IMMUNOPATHOGENESIS; MALE; ORAL DRUG ADMINISTRATION; PRIORITY JOURNAL; REBOUND; TOPICAL DRUG ADMINISTRATION;

EID: 0032994677     PISSN: 10811206     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1081-1206(10)62611-7     Document Type: Article

References (9)

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9. Aversa G, Punnonen J, Cocks BG, et al. An interleukin 4 (IL-4) mutant protein inhibits both IL-4 or IL-13-induced human immunoglobulin G4 (IgG4) and IgE synthesis and B cell proliferation: support for a common component shared by IL-4 and IL-13 receptors. J Exp Med 1993;178: 2213-18.