Li+ protects nerve cells against destabilization of Ca2+ homeostasis and delayed death caused by removal of external Na+

FEBS Letters

Volumn 448, Issue 1, 1999, Pages 173-176

  Khodorov, B ^a   Pinelis, V ^b   Vinskaya, N ^b   Sorokina, E ^b   Grigortsevich, N ^a   Storozhevykh, T ^b  

^a INSTITUTE OF GENERAL PATHOLOGY AND PATHOPHYSIOLOGY   (Russian Federation)

^b INSTITUTE OF BIOMEDICAL CHEMISTRY   (Russian Federation)

Author keywords

Cultured neuron; Glutamate; Lithium; Na+ Ca2+ exchange; Neurotoxicity

Indexed keywords

2 AMINO 5 PHOSPHONOVALERIC ACID; GLUTAMIC ACID; LITHIUM; MEGLUMINE; SODIUM;

ANIMAL CELL; ARTICLE; CALCIUM HOMEOSTASIS; CELL DEATH; CELL PROTECTION; CELL VIABILITY; CEREBELLUM; CONTROLLED STUDY; DELAYED NEUROTOXICITY; GRANULE CELL; NERVE CELL; NERVE CELL CULTURE; NEUROTOXICITY; NONHUMAN; PRIORITY JOURNAL; RAT; SODIUM CALCIUM EXCHANGE;

ANIMALS; CALCIUM; CELL DEATH; CELLS, CULTURED; HOMEOSTASIS; LITHIUM; N-METHYLASPARTATE; NEURONS; RATS; RATS, WISTAR; SODIUM;

EID: 0032994237     PISSN: 00145793     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0014-5793(99)00350-6     Document Type: Article

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