Liver AP-1 activation due to carbon tetrachloride is potentiated by 1,2- dibromoethane but is inhibited by α-tocopherol or gadolinium chloride

Free Radical Biology and Medicine

Volumn 26, Issue 9-10, 1999, Pages 1108-1116

  Camandola, Simonetta ^a   Aragno, Manuela ^a   Cutrin, Juan Carlos ^a   Tamagno, Elena ^a   Danni, Oliviero ^a   Chiarpotto, Elena ^a   Parola, Maurizio ^a   Leonarduzzi, Gabriella ^a   Biasi, Fiorella ^b   Poli, Giuseppe ^a,b  

^a UNIVERSITY OF TURIN   (Italy)

^b CNR   (Italy)

Author keywords

1,2 dibromoethane; AP 1; Carbon tetrachloride; Drug synergism; Free radical; Macrophage; tocopherol

Indexed keywords

1,2 DIBROMOETHANE; ALPHA TOCOPHEROL; CARBON TETRACHLORIDE; GADOLINIUM CHLORIDE; TRANSCRIPTION FACTOR AP 1;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ELECTROPHORETIC MOBILITY; LIPID PEROXIDATION; LIVER TOXICITY; MALE; NONHUMAN; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN DNA BINDING; RAT;

ANIMALIA;

EID: 0032982683     PISSN: 08915849     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0891-5849(98)00298-6     Document Type: Article

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