The role of Kir2.1 in the genesis of native cardiac inward-rectifier K+ currents during pre- and postnatal development

Annals of the New York Academy of Sciences

Volumn 868, Issue , 1999, Pages 434-437

  Nakamura, Tomoe Y ^a   Lee, Karen ^a   Artman, Michael ^a   Rudy, Bernardo ^a   Coetzee, William A ^a  

^a NEW YORK UNIVERSITY MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MESSENGER RNA; POTASSIUM CHANNEL; POTASSIUM ION;

ANIMAL CELL; CONFERENCE PAPER; CONTROLLED STUDY; FETUS; HEART VENTRICLE WALL; MEMBRANE STEADY POTENTIAL; MOUSE; NONHUMAN; POSTNATAL DEVELOPMENT; POTASSIUM CURRENT; PRENATAL DEVELOPMENT; TRANSCRIPTION REGULATION;

ANIMALIA;

EID: 0032969288     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1111/j.1749-6632.1999.tb11308.x     Document Type: Conference Paper

References (2)

1. KI with antisense oligonucleotides targeted to Kir2.1 mRNA. Am. J. Physiol. 274: H892-H900.
2. Hamill, O.P., A. Marty, E. Neher, B. Sakmann & F.J. Sigworth. 1981. Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patches. Pflug. Arch. Eur. J. Physiol. 391: 85-100.