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Volumn 11, Issue 3, 1999, Pages 889-898
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Episodic corticosterone treatment accelerates kindling epileptogenesis and triggers long-term changes in hippocampal CA1 cells, in the fully kindled state
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Author keywords
Calcium currents; Field potential; Glucocorticoid receptor; In situ hybridization; Mineralocorticoid receptor; Whole cell voltage clamp
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Indexed keywords
CALCIUM;
CORTICOSTEROID RECEPTOR;
CORTICOSTERONE;
GLUCOCORTICOID RECEPTOR;
MINERALOCORTICOID RECEPTOR;
RECEPTOR SUBTYPE;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
BRAIN NERVE CELL;
CALCIUM CURRENT;
CHANNEL GATING;
CONTROLLED STUDY;
ELECTRIC POTENTIAL;
EPILEPTOGENESIS;
HIPPOCAMPUS;
IN SITU HYBRIDIZATION;
KINDLING;
MALE;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
SUBCUTANEOUS DRUG ADMINISTRATION;
VOLTAGE CLAMP;
ACTION POTENTIALS;
ANIMALS;
ANTI-INFLAMMATORY AGENTS;
CALCIUM;
CORTICOSTERONE;
EPILEPSY;
EXCITATORY POSTSYNAPTIC POTENTIALS;
GENE EXPRESSION;
HIPPOCAMPUS;
IN SITU HYBRIDIZATION;
KINDLING, NEUROLOGIC;
MALE;
NEURONS;
PATCH-CLAMP TECHNIQUES;
RATS;
RATS, WISTAR;
RECEPTORS, GLUCOCORTICOID;
RECEPTORS, STEROID;
RNA, MESSENGER;
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EID: 0032967234
PISSN: 0953816X
EISSN: None
Source Type: Journal
DOI: 10.1046/j.1460-9568.1999.00495.x Document Type: Article |
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Times cited : (88)
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References (2)
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