Modification of a major peanut allergen leads to loss of IgE binding

International Archives of Allergy and Immunology

Volumn 118, Issue 2-4, 1999, Pages 313-314

  Burks, A Wesley ^a   King, Nina ^a   Bannon, Gary A ^a  

^a UNIVERSITY OF ARKANSAS FOR MEDICAL SCIENCES   (United States)

Author keywords

Hypoallergenic; IgE; Immunotherapy; Peanut allergen

Indexed keywords

ALLERGEN; COMPLEMENTARY DNA; IMMUNOGLOBULIN E;

ALLERGENICITY; AMINO ACID SUBSTITUTION; ANTIGEN BINDING; CONFERENCE PAPER; CONTROLLED STUDY; DNA SEQUENCE; HUMAN; HUMAN CELL; IMMUNOTHERAPY; LYMPHOCYTE PROLIFERATION; PEANUT; PRIORITY JOURNAL;

EID: 0032959071     PISSN: 10182438     EISSN: None     Source Type: Journal    
DOI: 10.1159/000024114     Document Type: Conference Paper

References (3)

1. Sampson HA: Food allergy. JAMA 1997;278: 1888-1894.
2. Burks AW, Williams LW, Connaughton C, Cockrell G, O'Brien T, Helm RM: Identification and characterization of a second major peanut allergen, Ara h II, utilizing the sera of patients with atopic dermatitis and positive peanuts challenge. J Allergy Clin Immunol 1992;90:962-969.
3. Stanley JS, King N, Burks AW, Huang SK, Sampson H, Cockrell G, Helm RM, West CM, Bannon GA: Identification and mutational analysis of the immunodominant IgE binding epitopes of the major peanut allergen Ara h 2. Arch Biochem Biophys 1997;3422:244-253.