|
Volumn 11, Issue 1, 1999, Pages 141-148
|
CaMKII-dependent phosphorylation of NR2A and NR2B is decreased in animals characterized by hippocampal damage and impaired LTP
a |
Author keywords
Hippocampus; Methylazoxymethanol; Postsynaptic densities; Rat; Synaptic plasticity
|
Indexed keywords
GLUTAMIC ACID;
METHYLAZOXYMETHANOL;
N METHYL DEXTRO ASPARTIC ACID RECEPTOR;
PHOSPHATASE;
PROTEIN KINASE (CALCIUM,CALMODULIN) II;
RECEPTOR SUBUNIT;
SERINE;
ANIMAL TISSUE;
ARTICLE;
AUTOPHOSPHORYLATION;
BRAIN CORTEX;
BRAIN INJURY;
CONTROLLED STUDY;
ENZYME ACTIVITY;
HIPPOCAMPUS;
LONG TERM POTENTIATION;
NERVE CELL PLASTICITY;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN PHOSPHORYLATION;
RAT;
SYNAPSE;
ANIMALS;
CA(2+)-CALMODULIN DEPENDENT PROTEIN KINASE;
CARCINOGENS;
ENZYME ACTIVATION;
FEMALE;
GENE EXPRESSION;
HIPPOCAMPUS;
IN SITU HYBRIDIZATION;
LONG-TERM POTENTIATION;
METHYLAZOXYMETHANOL ACETATE;
NERVE DEGENERATION;
NEURONS;
ORGAN CULTURE TECHNIQUES;
PHOSPHORYLATION;
PREGNANCY;
RATS;
RATS, SPRAGUE-DAWLEY;
RECEPTORS, N-METHYL-D-ASPARTATE;
RNA, MESSENGER;
SERINE;
SYNAPSES;
THREONINE;
TYROSINE;
|
EID: 0032945302
PISSN: 0953816X
EISSN: None
Source Type: Journal
DOI: 10.1046/j.1460-9568.1999.00414.x Document Type: Article |
Times cited : (30)
|
References (3)
|