Neutralization of endogenous NGF prevents the sensitization of nociceptors supplying inflamed skin

European Journal of Neuroscience

Volumn 11, Issue 5, 1999, Pages 1698-1704

  Koltzenburg, Martin ^a  

^a UNIVERSITY OF WÜRZBURG   (Germany)

Author keywords

Hyperalgesia; p75; Pain; Rat; trk

Indexed keywords

CARRAGEENAN; IMMUNOGLOBULIN G; NERVE GROWTH FACTOR; PROTEIN TYROSINE KINASE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; FEMALE; HEAT; HYPERALGESIA; MYELINATED NERVE; NOCICEPTIVE RECEPTOR; NONHUMAN; NONMYELINATED NERVE; PRIORITY JOURNAL; RAT; SENSORY NERVE; SKIN INFLAMMATION; SKIN NERVE; THERMAL STIMULATION;

ANIMALS; CARRAGEENAN; ELECTROPHYSIOLOGY; EXCIPIENTS; FEMALE; IMMUNOGLOBULIN G; NERVE GROWTH FACTORS; NEUROGENIC INFLAMMATION; NEURONS, AFFERENT; NOCICEPTORS; PHYSICAL STIMULATION; PROTO-ONCOGENE PROTEINS; RATS; RATS, WISTAR; RECEPTOR PROTEIN-TYROSINE KINASES; RECEPTOR, TRKA; RECEPTORS, NERVE GROWTH FACTOR; SKIN; STRESS, MECHANICAL;

EID: 0032906125     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1460-9568.1999.00590.x     Document Type: Article

References (1)

1. Abbadie, C., Trafton, J., Liu, H., Mantyh, P.W. & Basbaum, A.I. (1997) Inflammation increases the distribution of dorsal hom neurones that internalize the neurokinin-1 receptor in response to noxious and non-noxious stimulation. J, Neuroscience, 17, 8049-8060.